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GLP-1 treatment reduces endogenous insulin resistance via activation of central GLP-1 receptors in mice fed a high-fat diet
[14] [13] These mechanisms contribute to improved glycaemic control and weight loss, but they can also lead to reduced caloric intake, changes in eating patterns, and occasionally low blood glucose levels (hypoglycaemia), all of which may contribute to feelings of tiredness
However, based on their pharmacological profiles, the risk of direct interaction appears low because they operate on entirely separate receptor systems
Other experimental data confirm the effectiveness of ALC supplementation in protecting against brain damage, e.g., the application of 100 mg/kg body weight of ALC reduced the volume of brain injury and improved victims behavior after traumatic brain injury
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