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However, it can be manifested with the classical form of malarial fever, cerebral malaria, or malaria-induced anemia (MIA) and is responsible for one-third of malaria-associated deaths [401]

In addition, the absence of the P53 tumor suppressor gene can obstruct Gln utilization by decreasing GLS2 levels inside cells and boosting ROS levels ( 4 Targeting Gln to enhance clinical treatment of tumors The reprogramming of Gln metabolism is strongly linked to the aggressive development of tumors