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Clearly, increased alcohol consumption and fructose intake (particularly seen via ingestion of sweetened sodas and energy beverages sweetened with high-fructose corn syrup) promote increased uric acid production

While current therapies like sodium-glucose cotransporter 2 ( SGLT2 ) inhibitors and glucagon-like peptide-1 receptor agonists (GLP-1RAs) have improved outcomes, significant challenges persist in early detection and halting progression

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This observation led to the development of controlled cold-air chambers delivering temperatures below minus 110 degrees Celsius