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Excess copper is reduced by FDX1 to generate Cu, which drives cell death via two primary mechanisms: (1) Cu-mediated aggregation of lipoylated TCA cycle proteins (e.g., DLAT) and loss of Fe-S clusters, and (2) Cu-induced disruption of the Npl4/p97 protein quality-control system

This assumption is supported by the finding that only the overexpression of GCL, and not of GS, results in increased GSH levels (6)

Dermatologists explain the immune mechanism, why stress triggers it, how to identify it, and what treatments actually work for patchy hair loss