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Of the several possible hypotheses that have been advanced to explain this situation, one of the most plausible proposes that replication error is not responsible for the mutations causing for this disease

Importantly, differences in findings between ME/CFS and long COVID suggest that the underlying neurobiological mechanisms, while leading to similar clinical presentations, may differ

Plomin R, Fulker D, Corley R, DeFries JC (1997)

2 FcRn-Mediated Control of IgG and Albumin Homeostasis FcRn has a heterodimeric composition resembling a major histocompatibility complex (MHC) I molecule, with a unique transmembrane heavy chain (HC) that is paired with the common soluble 2-microglobulin (2m) [19]