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So, additional investigation is necessary to link these potential associations to konzo pathogenesis, particularly in relation to time of disease onset, as such markers may only be present during early phases of konzo onset when inflammation or neural damage is likely highest

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However, it has been recognized that the main cold defects at the cell wall level are primarily caused by the reduced solubilization and depolymerization of the pectin (Fruk et al., 2014

Increased insulin resistance was also shown in mice with diet-induced obesity and treated with Ralstonia pickettii , suggesting a potential role of this bacteria in T2D development [56]