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Use of the pharmacological inducer of ferroptosis, RSL3, elicited a similar level of lipid peroxidation in macrophages compared to H 2 O 2 , while the ferroptosis inhibitor, liproxstatin-1, significantly reduced the H 2 O 2 -induced cytotoxicity in macrophages

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The net result is a molecule that resists enzymatic breakdown considerably better than an unmodified sequence