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This attenuates angiotensin II (Ang II)-induced oxidative stress, DNA double-strand breaks, and podocyte damage, which is closely related to the pathogenesis of CKD [81] (Table 2)
However, IGF-1 DES is not without concernshypoglycemia risk, unknown long-term local tissue effects, and the fundamental question of whether truly site-specific muscle growth is achievable or desirable
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