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Memory traces Units of cognitive information in the brain that may cause structural or biochemical alterations allowing the storage of memory

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Mechanically, unregulated release of glutamate cannot be effectively buffered or cleared, resulting in impaired glutamate level in extracellular space, which may lead to activation of cell death pathways and secondary injury after acute TBI ( 2 O 2 -induced oxidative stress via increasing superoxide dismutase and glutathione peroxidase activities of bacteria, which may provide new insights of glutamic acid in cellular antioxidant activity ( Pgam5 -/- male mice) after TBI