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This highlights an important conceptual point: ferroptosis sensitivity is determined not only by GSH abundance but also by the metabolic capacity to sustain reductive flux under oxidative stress

The ATP-binding cassette sub-family B member 1 (ABCB1) is a membrane-standing ATP-dependent efflux pump defense mechanism against harmful substances, mediating docetaxel resistance by limiting intracellular drug concentrations [208,209]

Nance, K

Similarly, ferroptosis inhibitors prevent high-glucose-induced cardiomyocyte dysfunction in diabetes-induced cardiomyopathy, suggesting that ferroptosis may play an essential part in the development of diabetic cardiomyopathy [12]