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Additionally, prolonged L-dopa therapy is linked to folate, B6, and B12 deficiency (de Lau et al

This efficiency translates to more predictable therapeutic effects and potentially lower doses needed to achieve desired outcomes

Current research implicates several interrelated pathogenic mechanisms, including persistent neuroinflammation, mitochondrial dysfunction [27], excessive production of reactive oxygen species (ROS) [28], reduced dopamine synthesis [8], impaired cerebral glucose metabolism [8], and the accumulation of misfolded -synuclein in the form of Lewy bodies [29]

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