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Before nanocarrier cell entry experiments, the non-toxic concentrations of endocytosis and metabolic inhibitors on the cARLA treated hEC were determined by cell viability assays, namely impedance measurement and MTT tests (Fig
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Small human studies support the biological plausibility of both approaches, but there are no robust head-to-head clinical trials proving that one produces better health outcomes than the other
Initially EpRE was called the antioxidant response element (ARE) because the first compound, shown to activate ARE was a so-called antioxidant that was subsequently shown to generate H 2 O 2 through redox cycling (Pinkus et al., 1996)
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