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Thus, it is plausible that the hindbrain GLP-1 neurons are a part of the ascending pathway activated by intragastric glucose, with a role to limit reward responses perhaps

Evidence from clinical trials suggests that GLP-1 medicines exert clinical benefits even in individuals who do not experience substantial weight loss (25)

Once blood flow is restored, the reoxygenation of previously oxygen-deprived cells leads to dtissue damage called "reperfusion injury" which is accompanied by excessive generation of free radicals

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