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20 Therefore, the crosstalk among the gut, liver and brain is being increasingly recognized and delineated piece by piece (Fig

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While responses to incretin-based therapies vary across cancer types, analysis of existing data reveals several shared mechanisms that may explain their particular potential in obesity-associated cancers: i) Direct metabolic intervention: Tumor cells, particularly those evolving in an obese microenvironment, may exhibit heightened sensitivity to alterations in metabolic conditions

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