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Alterations in synaptic plasticity and oxidative stress following long-term paracetamol treatment in rat brain

Metabolized by proteolytic cleavage, -oxidation, and amide hydrolysis to constituent amino acids

These human disease conditions are mimicked by treatment of fasted C57Bl/6J mice with the standard overdose of 300 mg/kg APAP, which causes liver injury followed by regeneration and recovery without mortality