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Results demonstrated for the first time in the gastrointestinal that ionizing radiation persistently increases IGF1 and activates downstream PI3K/Akt and JAK2 signaling pathways, which may contribute to gastrointestinal functional alterations and carcinogenesis (Suman et al., 2015)

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While the two major types of diabetes, type 1 and type 2 (hereafter T1D and T2D, respectively), have their differences in pathogenesis, they share senescence of pancreatic -cells as a common pathogenetic mechanism 1,2